Thanks to lentiviral vectors, the authors investigate the role of UBE3C and ANXA7 in glioma. Their data reveal that high UBE3C expression contributes to glioma progression by ubiquitination and degradation of ANXA7, and thus presents a novel target for glioma therapy.
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Thanks to lentiviral vectors, the authors investigate the role of UBE3C and ANXA7 in glioma. Their data reveal that high UBE3C expression contributes to glioma progression by ubiquitination and degradation of ANXA7, and thus presents a novel target for glioma therapy.
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